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Teflex is a revolutionary new type of disinfectant, capable of ensuring
an environment free of pathogenic influence. The active ingredient
involved in Teflex's antimicrobial properties is a guanidine based
copolymer. The germicidal function is attributed to the ability of the
guanidine functional groups of the Teflex polymer bonding with the
cellular membranes of pathogenic microbes. Upon bonding, the guanidine
functional group infiltrates the cell's cytoplasm and inhibits the
cell's regulatory functions. These functions include: enzymatic
activity; the transfer of essential nutrients across the cell membrane;
cellular respiration; and the multiplication of the microbe. The
disruption of the cell's regulatory tasks is such that the cells can
no longer function the way that they need to in order to stay alive;
ultimately resulting in cell death.

The following is an abbreviated description of the destruction process
of the microorganisms:
- The absorption of the biocidal Teflex molecule through the microbes
cell membrane.
- The diffusion of the absorbed Teflex molecules through cell walls.
- The destabilization and destruction of the cytoplasmic membrane.
- The cell contents' disperse through the destabilized cell membrane,
leading to,
- The death of the cell.

The ability of Teflex to connect to and interfere with the cellular
membranes of microorganisms is attributed to the positively charged
ions in the quarter-amino functional groups of the Teflex polymer.
These positively charged sites of Teflex are attracted to the
negatively charged sites located in the pathogen's phospholipid
bilayer. It is essential to note that these negatively charged sites of
the microorganism's membrane are required in the composition of the
cellular membrane and its regulatory functions.
Thorough calculations indicate that each microbial cell reacts with
6x10^-8 (0.00000006) ml of the Teflex polymer.
Upon initial contact with the pathogen, there is an induction of
electro-static interference between the negatively charged groups on
the cellular membrane and the positively charged quarter-amino groups
of the Teflex polymer. This leads to a change in orientation in the
pathogen's membrane. The Teflex macromolecule connects with a large
number of phospholipids in the microbe's membrane; this reaction
takes place in a cooperative manner thereby evoking neutralization of
the negative charge in the cell's phospholipid bilayer. The resulting
compound of Teflex stabilizes, leading to an irreversible change in
electrostatic and hydrophobic interactions of the cell's membrane;
this results in further weakening of the membrane that is an essential
element to the life of the microbe. Eventually, lipid-lipid
interactions are weakened to a point where the membrane is so unstable
that the microbe no longer functions properly. Another result of the
Teflex fusion to the phospholipid bilayer is a break in the barrier and
transportation functions of the microbe's membrane.

As the Teflex polymer infiltrates even further, a hydrophobic fragment
penetrates into the non-polar part of the cellular membrane, leading to
a widening of the membrane and a disturbance of vanderwaal forces among
phospholipid molecules. This leads to permeability changes, followed by
a weakened membrane integrity, and eventually, the cell fragments and
dies.

All of these effects are directed against viruses, gram-positive and
gram-negative bacteria, fungi and protozoa; On the contrary, Teflex
does not harm the cells of larger organisms (i.e human or animal
cells). This is due to a difference in molecular structures and
biochemical properties, (for example, our cells contain cholesterol
which helps stabilize our membrane), and a difference in our cytoplasm
and nucleus.